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glutathione null Toxicogenomics of S-transferase (GST) gene family members: Chemical-gene interactions and potential implications of gene deletions Pulmonary disease
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glutathione null Toxicogenomics of S-transferase (GST) gene family members: Chemical-gene interactions and potential implications of gene deletions Pulmonary disease
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glutathione null Toxicogenomics of S-transferase (GST) gene family members: Chemical-gene interactions and potential implications of gene deletions Pulmonary disease
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glutathione null Toxicogenomics of S-transferase (GST) gene family members: Chemical-gene interactions and potential implications of gene deletions Pulmonary disease
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[DOI] [PMC free article] [PubMed] [Google Scholar] 336.Xue H., Hu L., Xiong Y., Zhu X., Wei C., Cao F., Zhou W., Sun Y., Endo Y., Liu M., et al
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They found that microbial infections, such as SARS and H5N1 virus infections in the lungs, trigger NADPH-dependent ROS production, thereby generating oxidized phospholipids, such as oxidized 1-palmitoyl-2-arachidonoyl-sn-glycerol-3-phosphatidylcholine, to induce lung injury and cytokine production by lung macrophages via TLR4–TRIF 80
Pulmonary disease
glutathione null Toxicogenomics of S-transferase (GST) gene family members: Chemical-gene interactions and potential implications of gene deletions Pulmonary disease
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