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Idiosyncratic Drug Reaction: A Rare Mechanism of Acute Tylenol Toxicity | Cureus
doi: 10.1074/jbc.M110.207738 82
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This upregulation promotes mitochondrial biogenesis, reduces apoptotic signaling, and inhibits inflammatory cascade responses, highlighting a mechanism through which HBO attenuates inflammation[68], By modulating these conserved cascades—notably via SIRT1 activation and regulation of apoptosis—HBO therapy contributes to improved functional recovery following central nervous system injuries, suggesting its potential to mitigate post-cardiac arrest encephalopathy by targeting shared inflammatory and apoptotic pathways in global cerebral ischemia[69, 70]
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